Arterial thrombosis is the formation of a thrombus within an artery.
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It also covers arterial thrombosis and embolism; vasculitides; and vasospastic disorders.
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Treatment of established arterial thrombosis includes the use of antiplatelet drugs and thrombolytic therapy.
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One death through coronary arterial thrombosis has been linked to ADB-FUBINACA intoxication.
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This is useful for the management of arterial thrombosis and prevention of adverse cardiovascular events.
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Antiplatelet drugs alter the platelet activation at the site of vascular damage crucial to the development of arterial thrombosis.
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The mechanism behind arterial thrombosis, such as with heart attacks, is more established than the steps that cause venous thrombosis.
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Excessive numbers of platelets, and / or normal platelets responding to abnormal vessel walls, can result in venous thrombosis and arterial thrombosis.
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Heparanase has been shown to promote arterial thrombosis and stent thrombosis in mouse models due to the cleavage of anti-coagulant heparan sulfate proteoglycans.
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With arterial thrombosis, blood vessel wall damage is required, as it initiates coagulation, but clotting in the veins mostly occurs without any such damage.